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cell lines hek 293t atcc crl 3216 molt4 ccr5 nih hiv reagent program arp  (ATCC)


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    ATCC cell lines hek 293t atcc crl 3216 molt4 ccr5 nih hiv reagent program arp
    Cell Lines Hek 293t Atcc Crl 3216 Molt4 Ccr5 Nih Hiv Reagent Program Arp, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 38849 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/cell lines hek 293t atcc crl 3216 molt4 ccr5 nih hiv reagent program arp/product/ATCC
    Average 99 stars, based on 38849 article reviews
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    (A–F, left) Jurkat, <t>MOLT4,</t> OCI-AML2, NALM-16, HCT-15, and SW480 cells were transduced with the indicated shRNAs, and the knockdown efficiency of SOD2 was assessed by RT-qPCR analysis in biological duplicates. (A–F, right) Jurkat, MOLT4, OCI-AML2, NALM-16 HCT-15, and SW480 cells were treated with the indicated doses of asparaginase in biological triplicates. After 8 days of treatment, relative viability was assessed. Cell counts were normalized to shLuc-transduced, vehicle-treated cells. (G–J) Jurkat cells were transduced with the indicated shRNAs and treated with the indicated doses of vincristine, dexamethasone, doxorubicin, and 6-mercaptopurine in biological duplicates. After 8 days of treatment, relative viability was assessed. Cell counts were normalized as in (A–F). All error bars represent SEM. *** p ≤ 0.001, ** p ≤ 0.01, * p < 0.05 by two-sided Student’s t test with Welch adjustment (A–F, left). See also and .
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    (A–F, left) Jurkat, <t>MOLT4,</t> OCI-AML2, NALM-16, HCT-15, and SW480 cells were transduced with the indicated shRNAs, and the knockdown efficiency of SOD2 was assessed by RT-qPCR analysis in biological duplicates. (A–F, right) Jurkat, MOLT4, OCI-AML2, NALM-16 HCT-15, and SW480 cells were treated with the indicated doses of asparaginase in biological triplicates. After 8 days of treatment, relative viability was assessed. Cell counts were normalized to shLuc-transduced, vehicle-treated cells. (G–J) Jurkat cells were transduced with the indicated shRNAs and treated with the indicated doses of vincristine, dexamethasone, doxorubicin, and 6-mercaptopurine in biological duplicates. After 8 days of treatment, relative viability was assessed. Cell counts were normalized as in (A–F). All error bars represent SEM. *** p ≤ 0.001, ** p ≤ 0.01, * p < 0.05 by two-sided Student’s t test with Welch adjustment (A–F, left). See also and .
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    Epizyme Inc molt4 cells
    (A–F, left) Jurkat, <t>MOLT4,</t> OCI-AML2, NALM-16, HCT-15, and SW480 cells were transduced with the indicated shRNAs, and the knockdown efficiency of SOD2 was assessed by RT-qPCR analysis in biological duplicates. (A–F, right) Jurkat, MOLT4, OCI-AML2, NALM-16 HCT-15, and SW480 cells were treated with the indicated doses of asparaginase in biological triplicates. After 8 days of treatment, relative viability was assessed. Cell counts were normalized to shLuc-transduced, vehicle-treated cells. (G–J) Jurkat cells were transduced with the indicated shRNAs and treated with the indicated doses of vincristine, dexamethasone, doxorubicin, and 6-mercaptopurine in biological duplicates. After 8 days of treatment, relative viability was assessed. Cell counts were normalized as in (A–F). All error bars represent SEM. *** p ≤ 0.001, ** p ≤ 0.01, * p < 0.05 by two-sided Student’s t test with Welch adjustment (A–F, left). See also and .
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    (A–F, left) Jurkat, MOLT4, OCI-AML2, NALM-16, HCT-15, and SW480 cells were transduced with the indicated shRNAs, and the knockdown efficiency of SOD2 was assessed by RT-qPCR analysis in biological duplicates. (A–F, right) Jurkat, MOLT4, OCI-AML2, NALM-16 HCT-15, and SW480 cells were treated with the indicated doses of asparaginase in biological triplicates. After 8 days of treatment, relative viability was assessed. Cell counts were normalized to shLuc-transduced, vehicle-treated cells. (G–J) Jurkat cells were transduced with the indicated shRNAs and treated with the indicated doses of vincristine, dexamethasone, doxorubicin, and 6-mercaptopurine in biological duplicates. After 8 days of treatment, relative viability was assessed. Cell counts were normalized as in (A–F). All error bars represent SEM. *** p ≤ 0.001, ** p ≤ 0.01, * p < 0.05 by two-sided Student’s t test with Welch adjustment (A–F, left). See also and .

    Journal: Cell reports

    Article Title: SOD2 is a regulator of proteasomal degradation promoting an adaptive cellular starvation response

    doi: 10.1016/j.celrep.2025.115434

    Figure Lengend Snippet: (A–F, left) Jurkat, MOLT4, OCI-AML2, NALM-16, HCT-15, and SW480 cells were transduced with the indicated shRNAs, and the knockdown efficiency of SOD2 was assessed by RT-qPCR analysis in biological duplicates. (A–F, right) Jurkat, MOLT4, OCI-AML2, NALM-16 HCT-15, and SW480 cells were treated with the indicated doses of asparaginase in biological triplicates. After 8 days of treatment, relative viability was assessed. Cell counts were normalized to shLuc-transduced, vehicle-treated cells. (G–J) Jurkat cells were transduced with the indicated shRNAs and treated with the indicated doses of vincristine, dexamethasone, doxorubicin, and 6-mercaptopurine in biological duplicates. After 8 days of treatment, relative viability was assessed. Cell counts were normalized as in (A–F). All error bars represent SEM. *** p ≤ 0.001, ** p ≤ 0.01, * p < 0.05 by two-sided Student’s t test with Welch adjustment (A–F, left). See also and .

    Article Snippet: MOLT4 cells , DSMZ , Cat#ACC-362.

    Techniques: Transduction, Knockdown, Quantitative RT-PCR